Formation of Fluorescent Pigment in Vitamin E Deficiency
نویسندگان
چکیده
منابع مشابه
Vitamin E Deficiency in the Monkey . Iv .
NEMIA regularly develops in the rhesus monkey (Macaca mulatta) when it is deprived of vitamin E.1 The anemia is accompanied by granulocytosis,’ an increased concentration of bone marrow deoxyribonucleic acid,2 a reduction in the maximal Cr5’ survival time of the erythrocytes,3 a slightly elevated reticulocyte count1 and a prompt reticulocytosis following vitamin E administration. This combinati...
متن کاملVitamin E Deficiency in the Monkey
Eight young rhesus monkeys were fed a purified diet devoid of vitamin E. After from 6 to 13 months of feeding, all the animals developed signs of vitamin E deficiency. The signs of vitamin E deficiency in the monkey include muscular dystrophy, elevated excretion of creatine, allantoin, and free amino acids and decreased excretion of creatinine. The vitamin E-deficient monkeys all developed anem...
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OBJECTIVE Ataxia with vitamin E deficiency (AVED) is a rare autosomal recessive neurological disorder which usually starts in childhood. The clinical presentation is very similar to Friedreich ataxia, most patients have progressive truncal and extremity ataxia, areflexia, positive Babinski sign, dysarthria and sensory neuropathy. METHODS We made an inquiry to our colleagues in Norway, we incl...
متن کاملsuppression of coke formation in thermal cracking by coke inhibitors
the main purpose of this research was to:1.develop a coking model for thermal cracking of naphtha.2.study coke inhibition methods using different coke inhibitors.developing a coking model in naphtha cracking reactors requires a suitable model of the thermal cracking reactor based on a reliable kinetic model.to obtain reliable results all these models shall be solved simultaneously.for this pu...
15 صفحه اولFormation of malonaldehyde in vitamin E deficiency and its relation to the inhibition of gulonolactone oxidase.
The impairment of synthesis in vitro of ascorbic acid by liver extracts from animals deprived of vitamin E (1, 2) is due to the inhibition of the enzyme(s) located in the microsomes (3) which catalyzes the oxidation of n-gulonolactone to L-ascorbic acid. This enzyme or enzymic complex will be termed gulonolactone oxidase. Concurrent with the inhibition of gulonolactone oxidase, a material is fo...
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ژورنال
عنوان ژورنال: British Journal of Nutrition
سال: 1947
ISSN: 0007-1145,1475-2662
DOI: 10.1079/bjn19470007